mutation research 投稿须知

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1、AUTHOR INFORMATION PACK 20 Jul MUTATION RESEARCH - FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS A section of Mutation Research - only available as part of a subscription to Mutation Research/Full Set AUTHOR INFORMATION PACK TABLE OF CONTENTS . XXX . Description Audience Impact Factor Abstract

2、ing and Indexing Editorial Board Guide for Authors p.1 p.1 p.1 p.2 p.2 p.4 ISSN: 0027-5107 DESCRIPTION . The scope of Mutation Research: Molecular and Fundamental Mechanisms broadly encompasses all aspects of research that address the detection of mutations, the mechanisms by which mutations in gene

3、s and chromosomes arise, and the modulation of mutagenesis by mutation avoidance pathways such as DNA repair, cell cycle control and apoptosis. It includes the role of genetic variation in the genesis and manifestation of mutations, ranging from the variable manner in which xenobiotics are metaboliz

4、ed to variations in the capacity of cells to replicate and repair damaged DNA. It also includes the contributions of these mechanisms, when perturbed, to animal disease models and to human disease, with particular emphasis on carcinogenic mechanisms. Chromosome stability is paramount for maintaining

5、 cellular homeostasis. Therefore, the Journal will publish articles on the genesis of aneuploidy and isodisomy, including the roles played by cell cycle checkpoints, spindle microtubules, centrosomes and kinetocore proteins, and agents that might disrupt them. Since isodisomy can occur as a conseque

6、nce of recombination, all aspects of homologous recombination and non-homologous end joining are appropriate. Submission of appropriate epidemiological studies as well as consequences, including methods for high throughput SNP detection, DNA microarrays and proteomic approaches, are welcome. The bro

7、ader scope of the journal is a reflection of the rapid advances in the field of mutation research and the recognition that cellular responses to DNA damage, including cell cycle checkpoint arrest and apoptosis, cannot be dissociated from the immediate mechanisms by which DNA is damaged and repaired.

8、 AUDIENCE . Mutageneticists, General Toxicologists, Molecular Geneticists, Cancer Researchers, Radiobiologists. IMPACT FACTOR . 2010: 3.204 Thomson Reuters Journal Citation Reports 2011 AUTHOR INFORMATION PACK 20 Jul ABSTRACTING AND INDEXING . BIOSIS Chemical Abstracts Current Contents/Life Sciences

9、 EMBASE EMBiology MEDLINE PASCAL M Reference Update Scopus EDITORIAL BOARD . EDITORS L.H.F. Mullenders, Toxicogenetica, Faculteit Geneeskunde, Divisie 5, Universiteit Leiden, Onderzoeksgebouw, Postbus 9600, 2300 RC Leiden, Netherlands, Fax: +31 71 5268284, Email: mutreslumc.nl P.J. Stambrook, Molecu

10、lar Genetics, Biochemistry for details you are referred to: Role of the funding source You are requested to identify who provided financial support for the conduct of the research and/or preparation of the article and to briefly describe the role of the sponsor(s), if any, in study design; in the co

11、llection, analysis and interpretation of data; in the writing of the report; and in the decision to submit the paper for publication. If the funding source(s) had no such involvement then this should be stated. Please see Funding body agreements and policies Elsevier has established agreements and d

12、eveloped policies to allow authors whose articles appear in journals published by Elsevier, to comply with potential manuscript archiving requirements as specified as conditions of their grant awards. To learn more about existing agreements and policies please visit US National Institutes of Health

13、(NIH) voluntary posting (“Public Access“) policy. AUTHOR INFORMATION PACK 20 Jul Special Subject Repositories Certain repositories such as PubMed Central ( PMC ) are authorized under special arrangement with Elsevier to process and post certain articles such as those funded by the National Institute

14、s of Health under its Public Access policy (see for more detail on our policy). Articles accepted for publication in an Elsevier journal from authors who have indicated that the underlying research reported in their articles was supported by an NIH grant will be sent by Elsevier to PMC for public ac

15、cess posting 12 months after final publication. The version of the article provided by Elsevier will include peer-review comments incorporated by the author into the article. Because the NIH Public Access policy is voluntary, authors may elect not to deposit such articles in PMC. If you wish to opt

16、out and not deposit to PMC, you may indicate this by sending an e-mail to: NIHauthorrequest Open access This journal offers you the option of making your article freely available to all via the ScienceDirect platform. To prevent any conflict of interest, you can only make this choice after receiving notification that your article has been accepted for publication. The fee of $3,000 excludes taxes and other potential author fees such as color char

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